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Methyltestosterone
Pharmaceutical Name: Methyltestosterone
Chemical structure: 17alpha-methyl-4-androstene-3-one,17b-ol
Effective dose: 25-50 mg/day
Methyltestosterone was, well, still is the worlds first oral steroid developed. Using the now infamous 17-alpha-methyl alteration to render the base hormone, testosterone, orally active. However, unlike the whole host of injectable testosterones, methyltest is a rather crude and not very well liked compound. Mostly due to this alteration. Methyltestosterone is to testosterone, what Dianabol (methandrostenolone) is to Equipoise (boldenone). On the one hand the 17-alpha-alkylation of the steroid gives it less affinity for the aromatase enzyme so less estrogen is formed, but as with Dianabol, the estrogen formed is 17-methyl-estradiol, which is much more potent. Just as we will notice serious bloat and water retention with Dianabol, we will see the same with methyltestosterone, but to a much greater degree, simply because the base structure has twice the tendency to aromatize. With this amount of estrogenic effects, gynocomastia is a very real threat and concomitant use of an anti-estrogen is strongly advised.
The alteration also decreases the affinity for other structures. First and foremost the androgen receptor. This offers us few benefits. Due to the decreased androgenic activity the potency of methyltestosterone is weaker than that of testosterone, but even in terms of androgenic risk nothing is really gained. Testosterone being the prime androgen, even with this alteration risk of hair loss, acne, prostate hypertrophy and a whole host of other side-effects is never far away. Also, where Dianabol has little to no conversion to a more active androgen by way of the 5-alpha-reductase enzyme, methyl-testosterone still shows fair affinity for this particular enzyme and converts to the powerful 17-methyl-Dihydrotestosterone. These type of side-effects alone will turn most experienced users off of methyl-testosterone, at least when equally priced and more controllable injectable products are available. As with any potent androgen, some men may develop aggressive tendencies during its use.
As with Dianabol, what we have on our hands here is a very potent mass builder and all in all an effective steroid when observed individually. 40-50 mg per day taken for just a few weeks can make drastic changes. But since many already find the bloat and fat gain of Dianabol a bit much to tolerate, this steroid is never in high demand. Dianabol is more available, provides extremely good results, is quite safe and comparatively cheap. So there is a multitude of reasons why methyltestosterone is rarely used. It seems, however, that it is making a re-introduction as a medical aid for oligospermic men, especially in the United States. One reason for this may in fact be the low demand for it on the black market, making more physicians comfortable in prescribing it due to a lowered chance of abuse.
Lastly, as with all 17-alpha-alkylated steroids, we need to mention the risk for liver damage. A methyl-testosterone product used for extensive time periods can cause severe hepatoxicity, so use is best limited to 6, maximum 8 weeks on end followed by an off-period of equal length or longer.
In conclusion, most will find methyl-testosterone to not be worth their while. The side-effects are ever present, and while they can easily be combated with a combination of arimidex and finasteride, it seems a bit idiotic to pay 15 or more dollars per day on ancillary drugs that will reduce the anabolic activity, while spending only 1-2 bucks at most on the steroid itself.
Those still seeking to use methyltest will probably do so out of necessity and will not be stacking it with another anabolic/androgenic steroid. For such use 40-50 mg taken in a single daily dose upon waking, for a period no longer than 8 weeks would be ideal. Some may wish to use this steroid, like Dianabol, to kickstart a cycle and get results sooner at the beginning of a longer cycle of injectable testosterone, possibly stacked with another base compound such as boldenone or nandrolone. In that case 30 mg or so, again in a single morning dose, taken for the first 5-6 weeks of said cycle would provide the needed benefits. Since this is only useful in bulking stacks with aromatizable steroids, the resulting severity of side-effects will be grave. One needs to verify he is not at risk for hair loss or prostate hypertrophy first, and have ancillary drugs such as Nolvadex, arimidex and finasteride on hand to control the side-effects.
In terms of ancillaries, If gynocomastia symptoms should appear, one should start the use of 20 mg of Nolvadex daily and start a cycle of 0.5 mg of anastrozole (arimidex) alongside it. After 3-4 days, the Nolva can be discontinued, but the anastrozole should be continued for a while longer. Some have asked me about the use of Proviron in this matter, but in my opinion one needs to realize how much DHT will be present with the use of this compound to begin with, it may do more harm than good to add more of it (Proviron being a 1-methyl-DHT). So granted, anastrozole is quite expensive, but needs to be given preference here. I don't normally approve of the use of finasteride, because DHT often offers a steroid user more benefits than problems (apart from those prone to hair loss) and the blocking thereof may reduce the results obtained, in this case, especially at the beginning of a longer injectable testosterone cycle, one may choose to look into its use. Natural testostosterone shutdown may be quite severe, so the use of HCG and Nolvadex or clomid post-cycle is virtually a must.
Pharmaceutical Name: Methyltestosterone
Chemical structure: 17alpha-methyl-4-androstene-3-one,17b-ol
Effective dose: 25-50 mg/day
Methyltestosterone was, well, still is the worlds first oral steroid developed. Using the now infamous 17-alpha-methyl alteration to render the base hormone, testosterone, orally active. However, unlike the whole host of injectable testosterones, methyltest is a rather crude and not very well liked compound. Mostly due to this alteration. Methyltestosterone is to testosterone, what Dianabol (methandrostenolone) is to Equipoise (boldenone). On the one hand the 17-alpha-alkylation of the steroid gives it less affinity for the aromatase enzyme so less estrogen is formed, but as with Dianabol, the estrogen formed is 17-methyl-estradiol, which is much more potent. Just as we will notice serious bloat and water retention with Dianabol, we will see the same with methyltestosterone, but to a much greater degree, simply because the base structure has twice the tendency to aromatize. With this amount of estrogenic effects, gynocomastia is a very real threat and concomitant use of an anti-estrogen is strongly advised.
The alteration also decreases the affinity for other structures. First and foremost the androgen receptor. This offers us few benefits. Due to the decreased androgenic activity the potency of methyltestosterone is weaker than that of testosterone, but even in terms of androgenic risk nothing is really gained. Testosterone being the prime androgen, even with this alteration risk of hair loss, acne, prostate hypertrophy and a whole host of other side-effects is never far away. Also, where Dianabol has little to no conversion to a more active androgen by way of the 5-alpha-reductase enzyme, methyl-testosterone still shows fair affinity for this particular enzyme and converts to the powerful 17-methyl-Dihydrotestosterone. These type of side-effects alone will turn most experienced users off of methyl-testosterone, at least when equally priced and more controllable injectable products are available. As with any potent androgen, some men may develop aggressive tendencies during its use.
As with Dianabol, what we have on our hands here is a very potent mass builder and all in all an effective steroid when observed individually. 40-50 mg per day taken for just a few weeks can make drastic changes. But since many already find the bloat and fat gain of Dianabol a bit much to tolerate, this steroid is never in high demand. Dianabol is more available, provides extremely good results, is quite safe and comparatively cheap. So there is a multitude of reasons why methyltestosterone is rarely used. It seems, however, that it is making a re-introduction as a medical aid for oligospermic men, especially in the United States. One reason for this may in fact be the low demand for it on the black market, making more physicians comfortable in prescribing it due to a lowered chance of abuse.
Lastly, as with all 17-alpha-alkylated steroids, we need to mention the risk for liver damage. A methyl-testosterone product used for extensive time periods can cause severe hepatoxicity, so use is best limited to 6, maximum 8 weeks on end followed by an off-period of equal length or longer.
In conclusion, most will find methyl-testosterone to not be worth their while. The side-effects are ever present, and while they can easily be combated with a combination of arimidex and finasteride, it seems a bit idiotic to pay 15 or more dollars per day on ancillary drugs that will reduce the anabolic activity, while spending only 1-2 bucks at most on the steroid itself.
Those still seeking to use methyltest will probably do so out of necessity and will not be stacking it with another anabolic/androgenic steroid. For such use 40-50 mg taken in a single daily dose upon waking, for a period no longer than 8 weeks would be ideal. Some may wish to use this steroid, like Dianabol, to kickstart a cycle and get results sooner at the beginning of a longer cycle of injectable testosterone, possibly stacked with another base compound such as boldenone or nandrolone. In that case 30 mg or so, again in a single morning dose, taken for the first 5-6 weeks of said cycle would provide the needed benefits. Since this is only useful in bulking stacks with aromatizable steroids, the resulting severity of side-effects will be grave. One needs to verify he is not at risk for hair loss or prostate hypertrophy first, and have ancillary drugs such as Nolvadex, arimidex and finasteride on hand to control the side-effects.
In terms of ancillaries, If gynocomastia symptoms should appear, one should start the use of 20 mg of Nolvadex daily and start a cycle of 0.5 mg of anastrozole (arimidex) alongside it. After 3-4 days, the Nolva can be discontinued, but the anastrozole should be continued for a while longer. Some have asked me about the use of Proviron in this matter, but in my opinion one needs to realize how much DHT will be present with the use of this compound to begin with, it may do more harm than good to add more of it (Proviron being a 1-methyl-DHT). So granted, anastrozole is quite expensive, but needs to be given preference here. I don't normally approve of the use of finasteride, because DHT often offers a steroid user more benefits than problems (apart from those prone to hair loss) and the blocking thereof may reduce the results obtained, in this case, especially at the beginning of a longer injectable testosterone cycle, one may choose to look into its use. Natural testostosterone shutdown may be quite severe, so the use of HCG and Nolvadex or clomid post-cycle is virtually a must.
