Miotolan

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Miotolan
Pharmaceutical Name: Furazabol
Chemical structure: 17-alpha-methyl-5-alpha-androsta-2,3-furazan,17b-ol
Effective dose: 20-50 mg/day

Furazabol reminds us of Stanozolol (Winstrol) strucrurally. Its similar in appearance in that it's a DHT molecule with a 17-alpha-methyl group for oral availability, and has no 3-keto group, needed for androgenic binding. But instead of a 2,3-pyrazol group, furazabol has a 2,3-furazan group. The difference may not be all that big, both groups contain 2 nitrogen atoms and 2 double bonds and both are present instead of the 3-keto group. The advantage is that its not readily deactivated and therefore whatever influences it has, they are consistent. The downside is that the lack of a 3-keto group, which will impair its overall androgenic potency. So in that aspect again comparable to stanozolol. Anabolics 2002, without a doubt the best reference guide for steroids in print, lists Furazabol as extremely androgenic however, which is no doubt just an oversight. In nearly every way the behaviour of furazabol would be identical to that of Stanozolol.

It's an obscure steroid, that's the least we can say. Its only manufactured in Japan and in tabs of 1 mg. Low availability makes the cost of this steroid rather high, and its not particularly easy to find. Perhaps a tad more potent than Stanozolol, the doses used lay in the same neighbourhood, 20-50 mg/day. The higher doses being the preference. The demand for it isn't very high either, because Winstrol/Stromba is a popular and cheap to come by. The only benefit of its obscurity is that noone will invest in faking it. So if you do come across Furazabol, you have pretty good odds that the stuff is legit.

Now, the literature does not make a whole lot of mention of furazabol, but from what I was able to find1, it supports the weak nature of the steroid. In one case it was found that furazabol was a good treatment for hyperlipemia, and this without affecting proteinuria (the prevention of excretion of amino acids, where one would expect a steroid to increase proteinuria and not effect hyperlipemia). The low androgen binding may explain the lack of effect it had on proteinuria. The doses used were considerably high though, at least for furazabol. 1.1 mg/kg/day. That means a 200 lb bodybuilder would be using around 90-100 mg/day

Furazabol can be considered a relatively light steroid therefore. It is not estrogenic in anyway, on account of its dihydro structure and its lack of estrogenic action and low androgenic binding make it have fairly little influence on the body's own testosterone production. Much like Winstrol (stanozolol) and Anavar (oxandrolone). In the long run suppression will occur of course, but because it occurs much slower a user will suffer less from testicular atrophy and therefore bounce back easier when a cycle is concluded. There is a slim chance of androgenic risk, as with Winstrol, but its not frequent or severe. So acne, increased body and facial hair and even an aggravation of male pattern hair loss can occur, but it's a lot less likely than with more androgenic specimen.

Furazabol is a 17-alpha-alkylated steroid, and therefore has a level of hepatoxicity. In the interest of protecting your liver, you should not extend use beyond 6-8 weeks maximum. It's a mild steroid with no estrogenic activity, so logically its best used when cutting in stacks with Equipoise (boldenone undecylenate), Finaplix (trenbolone acetate) or Primobolan (methenolone enanthate) and the needed fat-burners of course. Unlike most steroids, this drug has a relatively short half-life2 however. It compensates with quite long activity (15-33% excretion of unchanged metabolites after 24 hours) so a single dose should be enough to get you through the day. But on account of the low half-life time, you may want to consider splitting doses in two each day. Because it doesn't aromatize and doesn't have a strong androgenic component, the use of ancillary drugs is limited. The use of Clomid or Nolvadex after a cycle is certainly advised, though the merit may be rather limited. There is no need for anti-estrogens or blood pressure medication during the cycle.

Orabolin
Pharmaceutical Name: ethylestrenol
Chemical structure: 19-Nor-17alpha-pregn-4-en-17-ol
Effective dose: 20-50 mg/day

People who have been in this game for a long time, may remember this steroid as Maxibolin. Its most popular name. But to avoid all confusion, its now best referred to as Orabolin, because Maxibolan was taken from the market some time ago. Most experienced users will of course prefer not to remember Orabolin at all. It was a bit of a failure in all aspects.

First of all it's a 19Nor-steroid, a derivative of nandrolone. I'm no big fan of 19Nor-steroids, apart from maybe trenbolone. The lack of the 19th carbon makes them re-esterify easily, particularly suppressive of natural testosterone and above all, lends them progestagenic activity, or if you will, the ability to worsen estrogenic side-effects by binding the progesterone receptor. The sole benefit of a 19nor compound is that it has very good androgen binding properties, giving it good enough anabolic effect, but is actually androgenically reduced in androgen responsive tissues like prostate and skin. This allowed users to book decent gains without overly having to fear acne, hair loss and prostate hypertrophy as they did with testosterone. For me that still doesn't warrant the use of nasty stuff like nandrolone or norethandrolone, for some it does. But I'm pretty sure all will be in agreement that this steroid is a waste. Its similar to norethandrolone, except it lacks a 3-keto group. This group is essential for binding the androgen receptor, and without it, its safe to assume that the anabolic activity of this steroid is less than weak. Studies1 actually seem to suggest that the only anabolic activity that ethylestronol does exert, is by making a 3-keto group and thus converting to norethandrolone.

Norethandrolone (inviting you to read the profile on norethandrolone) wasn't much of a success either. It was designed to be an androgenically mild oral steroid, like an oral nandrolone (which is in essence what it was), but then Searle realized that apart from being androgenically mild it was relatively nasty and uncontrollable stuff (which is how I feel about most 19nor steroids, including nandrolone) and came out with Anavar instead, since it was better suited as a mild oral steroid. So even through conversion you don't get anything decent out of this product.

The 17-alpha-ethyl group also lends it a certain liver toxicity, which doesn't allow for long use or high doses. And high doses are really what you need for any form of favorable effect. Women may somewhat appreciate this steroid in doses of 20-30 mg day, as its androgenically the mildest you'll ever come across, with very little virilizing symptoms. Although in any case, I would still recommend Anavar (oxandrolone) over ethylestrenol. Mainly due to its reputation, its become hard to find on the black market. Virtually extinct. I for one don't really care much.If you have ethylestrenol, my advice is toss it or sell it to your gullable friend. It's a waste. If you must use its, remember that the gains will be next to non-existent. Using 30-50 mg per day for 5-6 weeks on end, stacked with other products are the best way to go. Make it worth your while and add in some testosterone or boldenone for example. Aromatization is minimal, so the use of anti-estrogens will not be needed during the cycle, but because 19Nor steroids are nasty, suppressive stuff, you would do wise to run HCG and Nolvadex or Clomid once the cycle is over.
 
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