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08-13-2014, 01:42 AM
AICAR and EnzymesScientific study based on animal test subjects has been able to hone in on AICAR’s ability to enter cardiac cells and prohibit the creation of two specific enzymes; adenosine kinase (ADK), and adenosine deaminase (ADA). The first enzyme serves to catalyze the transfer of the gamma phosphate from ATP to adenosine, which then serves as a controller of aggregations of both extracellular and intracellular adenine nucleotides. The second enzyme breaks down adenosine which then is responsible for the turnover in nucleic acid in tissue. Because these enzymes are blocked, AICAR allows for an accelerated stimulation of glucose uptake and an increase in the activity of p38 mitogen-activated protein kinases α and β in skeletal muscle tissue. These two processes combine to form an accelerated process of energy conversion. Additionally, the peptide has been known to suppress the process of programmed cell death by lowering the production of oxygen compounds that are found within the cellular unit.

AICAR and Boosted Energy Regulation
According to scientific study based on animal test subjects, it has been determined that AICAR can play a vital role in boosting the regulation of energy.
The first boost tied to the peptide is based on its ability to boost the flow of blood through an animal test subject’s circulatory system in order to provide energy more efficiently. Because of this process, it is though that animal test subjects can function at an elevated level of activity over a longer interval of time before degradation brought upon by fatigue begins to manifest.
The second boost that is associated with the peptide is linked to the way in which AICAR is associated with adipose tissue, also known as body fat. Because AICAR has been shown to provide acceleration to an animal test subject’s metabolic processes, it has been determined through scientific study that the presence of the peptide can cause the breaking down of fat cells on a much quicker basis. This process is done as a means of compensation for the boost in metabolism. That said, it has been determined that this accelerated breaking down of body fat only occurs when an animal test subject stays on a regulated diet and does not experience a boosted intake of food.