M1T

doozer said:

ok now i testosterone igf-1 long r get it i try putiing this in order rather satelite coll regulation through cell regulation ocurs when onther muscle works harder for pseudohypertrophoy with of the many grow facters thyamidine bromodeoxyyuridne desmin myod1 moc first muscle specific perteins myogonosis ans is enhanced by steroid use the incorporatinting into existing fibers and to maintain nuclear cytoplasmic ratio is a fundamental mechanism fo muscle growth atrophine inhibits the actions acetylcholine on exocrine and smooth muscle but not on skeletal muscle (many diffrent drugs or recongition sites) or termed recepters or hormone reecpters are identified by there structures and for th perfomances they do B-andregenic subtypes benzodiazpins and the insulin family to there contemplating recepters in vito can reveal important aspacts of there behavior subcellur cellular when taking tesosterone for long periods of time lead to down regulation pottassium k psuedohyperkalema hypoglycymia in the pressence of insulin defiancy moderatly moderatley heavey ecersize perticualy in the B-blockers steroid anti inflamatory drugs now we down whant that messing up our gains possibly enough vitamin b 12 kynosolon keep up with water electrolytes minerals this will be very wise gh is liberated through protoelclyte attivity anitiatiate an ativaion process where prolife active phase after myofibers are formed the cell behavior is required through difusing features arising from the fibers that just would not be a good thing adrenalcortorpol horomone (ACTH)is a simple poly peptide containing 39 amino acids biological n terminal 20 amino acids CRH is the primary agent that releases (ACTH:arcade: :arcade: :arcade:

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doozer said:

don t u guys under stand

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