Anabolic and androgenic

I always thought that anabolic meant growth and androgenic meant sexual changes such as deepening of the voice and pubic hair growth.

I read that one should use more anabolic agents at the beginning of a cycle and more androgenic compounds at the end. What exactly is the purpose of doing this and is there another meaning to androgenic that I am overlooking? Any clarification would be great! Thanks

Here are the defenitions.

Anabolic: Pertaining to anabolism; an anabolic changes, or processes, more or less constructive in their nature.

Anabolism: The phase of metabolism in which simple substances are synthesized into the complex materials of living tissue.

Androgenic: of or related to the male hormone androgen

Androgen: A steroid hormone, such as testosterone or androsterone, that controls the development and maintenance of masculine characteristics. Also called androgenic hormone.

I am not familiar with this method of using more androgenic hormones at the end of a cycle, maybe it help retain muscle? or maybe its someones BS?

Thanks Cordoba! Yeah they talked about using halo, masteron, winny, tren, proviron, even dbol at the end of a cycle.

Here is some more info from the post;
Originally Posted by big k.l.g
Hi bros,
First most of us know that Androgen Receptor sites do not "burn out" this is hogwash in fact the opposite is true, the AR upregulate and muliply to make use of all the new high concentrations in blood....the reason you need to increace AS doses after a few cycles is because of the added muscle mass ( more AR sites) from previous cycle. NOW how do i keep on growing during a say 20 week cycle? Assuming diet, rest, training etc is good is there anything that can be done to maximize AAS caused growth? the one i know that slows down cycle muscle gains of is the SHBG increase, which i can combat with the use of proviron+insulin. Are there any other growth-slowing actions i should know about? thanks guys.


einstein1905:

I've posted my theory on AR receptors over prolonged exposure to supraphysiological doses of AAS over time. ARs are upregulated initially in response to increased ligand (AAS) concentrations. However, it's been shown in vitro (but not yet in vivo) that ARs do downregulate over time, when exposed to constant high levels of AAS (test). Androgens lower SHBG levels, whereas estrogens increase SHBG levels, so utilizing sufficient anti e's throughout a cycle can have advantages from many different standpoints. During an AAS cycle, if sufficient anti e's are used, your bioavailable AAS (akin to bioavailable test in a "natural" athlete) will be much higher than when not on cycle. SHBG will creep up over time, but your % bioavailable test should still be much higher than off cycle. A couple things to try during the latter half or latter 1/3 of a longer cycle would be to incorporate proviron and/or to slightly increase doses (but plan your starting doses with this in mind). I'm in no way a proponent of big doses. Your body seeks homeostasis by many mechanisms and will adjust to Xmg/wk of AAS. Increasing that dose after a long run at a constant dose has the potential to increase expression of ARs once again. Another thing that many do is to add in more androgenic compounds towards the end of their longer cycles, which can again lower SHBG levels in addition to providing their inherent anabolic and androgenic effects.

Originally Posted by TheMudMan

Also, another thing I have learned is when gains stop comming on it's ususally because your calorie intake needs to be bumped up.........

einstein1905:

This can't be stressed enough. I should have mentioned that before anything else I said. You NEED to maintain a fairly excessive positive caloric intake to grow big.



Over the course of a longer cycle, you'll have sustained elevated estrogen levels for many weeks (which is beneficial in and of itself), but it also increases SHBG, which binds AAS to result in a lower % of AAS molecules free to carry out their effects in the body. Androgenic compounds lower SHBG, so adding androgenic compounds at the end not only adds their inherent effects but also reduces SHBG to amplify the effects of the other AAS in use